Oxidative Stress: At the cellular level, it causes disruptions in intracellular organelles and functions (cell membranes, mitochondria; ATP production, ribosomes; protein synthesis), intracellular signaling systems, DNA/RNA structures, DNA methylation pathways, and epigenetic changes. The main oxidative stress molecules are reactive oxygen species (ROS), reactive nitrogen species (RNS), superoxide (O₂⁻), and hydroxyl radicals (HO•).

Glutathione (L-γ-glutamyl-L-cysteinyl-glycine) is a tripeptide composed of three amino acids: glutamine, cysteine, and glycine.

Liposomal Glutathione: Antioxidant therapy prevents oxidative stress and, if activated, controls it. One of the most effective molecules in antioxidant therapy is Liposomal Glutathione.

In autism (ASD) there is both inflammation (neuroinflammation) and oxidative stress activation in nerve cells in the brain, and as a result, an increase in the number of microglial cells and the presence of functional disorders have been reported. Antioxidant therapy in autism shows very significant and critical positive effects on controlling oxidative stress damage and neuronal inflammation in nerve cells (neurons) in the brain, as well as controlling the increase in the number of microglial cells and functional disorders as a result. These protective effects at the cellular and molecular levels can lead to positive results in preserving the vitality of nerve cells in medical terms, maintaining neuronal signaling systems, regulating synaptic pruning and the synaptic architectural structure, and ensuring healthy organization and functioning of the neuronal network. The repairs and adjustments in the structure and functions of nerve cells, synaptic structures, and microglial cells in the brain are directly related to the reduction and improvement of behavioral disorders.